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E2 increases the potency of CCK by increasing the sensitivity of vagal CCK-A receptors, although it does not increase CCK secretion or the number of CCK-A receptors (84–87)
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Results from a study involving a small number of patients showed that the overall risk of gastrointestinal or surgical site bleeding associated with intravenous KTR treatment was only slightly higher than for other sensitivities to this drug
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