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For connective tissue, Tβ4 operates through matrix metalloproteinase (MMP) inhibition — a distinct mechanism from BPC-157's VEGFR2 pathway — reducing enzymatic degradation of collagen, fibronectin, and laminin in arthritic joints, degenerative discs, and aging ligamentous tissue
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Beyond its prominent status in obesity trials, a significant branch of active research explores AOD9604’s impact on cartilage tissue and joint wear models
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