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Thymosin Beta 4 Actin-Sequestering Mechanism Thymosin Beta 4 functions as the principal G-actin sequestering peptide in the cytoplasm, binding monomeric actin in a 1:1 complex that inhibits its polymerization into filaments, a mechanism central to laboratory research on cell motility and differentiation
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10.1016/S0140-6736(19)31775-1 Lancet
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