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In some embodiments, the invention encompasses contacting one or more NNMT inhibitors (such as a cation of Formula I or IA, or otherwise disclosed herein) is contacted with a cell expressing NNMT concurrently with one or more chemical entities (e.g., nicotinamide riboside, nicotinamide mononucleotide) that modulate intracellular NAD+ levels to provide muscular therapy

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Originally developed for the treatment of type 2 diabetes through it’s action as a GLP-1 receptor agonist, which causes increased insulin secretion and decreased glucagon secretion by the pancreas in response to glucose (sugar) exposure (i.e