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DDIT3 promotes glycolysis by inhibiting TP53-induced glycolysis and apoptosis regulator (TIGAR) to generate ATP, while simultaneously facilitating the degradation of mitochondrial electron transport chain proteins [such as coenzyme Q9 homolog (COQ9) and cytochrome c oxidase subunit 4 (COX4)] via lon peptidase 1, mitochondrial (LONP1), thereby suppressing oxidative phosphorylation and reducing reactive oxygen species (ROS) production
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