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[5] It has been hypothesized to act through GH-mediated pathways on the osteoblasts (bone-forming cells), possibly leading to increased proliferation, development, and differentiation of the osteoblasts
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Further study of VEGFR-2, its related pathways, and the interaction of biochemical and biomechanical signaling that control vascular growth will aid researchers in developing new treatments that more efficiently treat the underlying cause of various disease states
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