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To identify the kinases potentially responsible for GRIP1 phosphorylation in macrophages, we tested a panel of kinase inhibitors and found that flavopiridol (FVP), a CDK9 inhibitor, abrogated dexamethasone-dependent GRIP1 phosphorylation in both THP1 MΦ and BMMΦ (Fig

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M2 macrophage polarization is driven by IL-4 and IL-13 signals through a heterodimeric receptor complex composed of the IL-4 receptor alpha chain (IL-4Rα) and the IL-13 receptor alpha 1 chain (IL-13Rα1) (90, 91) ( Figure 1B )