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In addition, it might counteract the dopamine inhibition (i.e., haloperidol-induced catalepsy), destruction and depletion (i.e., motor abnormalities induced by parkinsongenic neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) or reserpine) and over-stimulation (i.e., the adverse effects of the acute and chronic amphetamine, acute apomorphine, chronic methamphetamine) (for review, see [8,9,10])

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moreover, the permeability of the TJ depends largely on the claudin distribution pattern and the interactions between the claudins